anti wisp1 antibodies (Santa Cruz Biotechnology)
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Santa Cruz Biotechnology
anti wisp1 antibodies
Anti Wisp1 Antibodies, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 33 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+wisp1+antibodies/WISP-1+Antibody/pm39468941-57-31-33
Average 93 stars, based on 33 article reviews
Anti Wisp1 Antibodies, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 33 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+wisp1+antibodies/WISP-1+Antibody/pm39468941-57-31-33
Average 93 stars, based on 33 article reviews
anti wisp1 antibodies - by Bioz Stars,
2026-09
93/100 stars
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Protease Inhibitor:Article Title: The carcinogenesis of esophageal squamous cell cancer is positively regulated by USP13 through WISP1 deubiquitination. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 82373010; Science and Technology Innovation Plan Of Shanghai Science and Technology Commission, Grant/Award Number: 21ZR1411200 Abstract The objective was to determine whether USP13 stabilizes WISP1 protein and contributes to tumorigenicity and metastasis in ESCC through the Wnt/CTNNB1 signaling pathway.. ESCC cell lines (KYSE150 and TE10) were treated with the proteasome inhibitor MG-132, followed by siRNA screening of deubiquitinases (DUBs) to identify regulators of WISP1.. Mass spectrometry, immunoprecipitation, and in vitro functional assays were conducted to explore the interaction between USP13 and WISP1 and to assess the effects of USP13 downregulation on cell proliferation, migration, invasion, epithelial– mesenchymal transition (EMT), and apoptosis. Incubation:Article Title: The carcinogenesis of esophageal squamous cell cancer is positively regulated by USP13 through WISP1 deubiquitination. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 82373010; Science and Technology Innovation Plan Of Shanghai Science and Technology Commission, Grant/Award Number: 21ZR1411200 Abstract The objective was to determine whether USP13 stabilizes WISP1 protein and contributes to tumorigenicity and metastasis in ESCC through the Wnt/CTNNB1 signaling pathway.. ESCC cell lines (KYSE150 and TE10) were treated with the proteasome inhibitor MG-132, followed by siRNA screening of deubiquitinases (DUBs) to identify regulators of WISP1.. Mass spectrometry, immunoprecipitation, and in vitro functional assays were conducted to explore the interaction between USP13 and WISP1 and to assess the effects of USP13 downregulation on cell proliferation, migration, invasion, epithelial– mesenchymal transition (EMT), and apoptosis. Magnetic Beads:Article Title: The carcinogenesis of esophageal squamous cell cancer is positively regulated by USP13 through WISP1 deubiquitination. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 82373010; Science and Technology Innovation Plan Of Shanghai Science and Technology Commission, Grant/Award Number: 21ZR1411200 Abstract The objective was to determine whether USP13 stabilizes WISP1 protein and contributes to tumorigenicity and metastasis in ESCC through the Wnt/CTNNB1 signaling pathway.. ESCC cell lines (KYSE150 and TE10) were treated with the proteasome inhibitor MG-132, followed by siRNA screening of deubiquitinases (DUBs) to identify regulators of WISP1.. Mass spectrometry, immunoprecipitation, and in vitro functional assays were conducted to explore the interaction between USP13 and WISP1 and to assess the effects of USP13 downregulation on cell proliferation, migration, invasion, epithelial– mesenchymal transition (EMT), and apoptosis. Immunoprecipitation:Article Title: The carcinogenesis of esophageal squamous cell cancer is positively regulated by USP13 through WISP1 deubiquitination. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 82373010; Science and Technology Innovation Plan Of Shanghai Science and Technology Commission, Grant/Award Number: 21ZR1411200 Abstract The objective was to determine whether USP13 stabilizes WISP1 protein and contributes to tumorigenicity and metastasis in ESCC through the Wnt/CTNNB1 signaling pathway.. ESCC cell lines (KYSE150 and TE10) were treated with the proteasome inhibitor MG-132, followed by siRNA screening of deubiquitinases (DUBs) to identify regulators of WISP1.. Mass spectrometry, immunoprecipitation, and in vitro functional assays were conducted to explore the interaction between USP13 and WISP1 and to assess the effects of USP13 downregulation on cell proliferation, migration, invasion, epithelial– mesenchymal transition (EMT), and apoptosis. SDS Page:Article Title: The carcinogenesis of esophageal squamous cell cancer is positively regulated by USP13 through WISP1 deubiquitination. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 82373010; Science and Technology Innovation Plan Of Shanghai Science and Technology Commission, Grant/Award Number: 21ZR1411200 Abstract The objective was to determine whether USP13 stabilizes WISP1 protein and contributes to tumorigenicity and metastasis in ESCC through the Wnt/CTNNB1 signaling pathway.. ESCC cell lines (KYSE150 and TE10) were treated with the proteasome inhibitor MG-132, followed by siRNA screening of deubiquitinases (DUBs) to identify regulators of WISP1.. Mass spectrometry, immunoprecipitation, and in vitro functional assays were conducted to explore the interaction between USP13 and WISP1 and to assess the effects of USP13 downregulation on cell proliferation, migration, invasion, epithelial– mesenchymal transition (EMT), and apoptosis. Western Blot:Article Title: The carcinogenesis of esophageal squamous cell cancer is positively regulated by USP13 through WISP1 deubiquitination. Article Snippet: Funding information National Natural Science Foundation of China, Grant/Award Number: 82373010; Science and Technology Innovation Plan Of Shanghai Science and Technology Commission, Grant/Award Number: 21ZR1411200 Abstract The objective was to determine whether USP13 stabilizes WISP1 protein and contributes to tumorigenicity and metastasis in ESCC through the Wnt/CTNNB1 signaling pathway.. ESCC cell lines (KYSE150 and TE10) were treated with the proteasome inhibitor MG-132, followed by siRNA screening of deubiquitinases (DUBs) to identify regulators of WISP1.. Mass spectrometry, immunoprecipitation, and in vitro functional assays were conducted to explore the interaction between USP13 and WISP1 and to assess the effects of USP13 downregulation on cell proliferation, migration, invasion, epithelial– mesenchymal transition (EMT), and apoptosis. |
